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Senescence Reprogramming by TIMP1 Deficiency Promotes Prostate Cancer Metastasis

Developmentally-programmed cellular senescence is conserved and widespread in zebrafish

The bright and dark side of extracellular vesicles in the senescence-associated secretory phenotype

T cells with dysfunctional mitochondria induce multimorbidity and premature senescence

Global hyperactivation of enhancers stabilizes human and mouse naive pluripotency through inhibition of CDK8/19 Mediator kinases

H3K9me3-mediated epigenetic regulation of senescence in mice predicts outcome of lymphoma patients

AP-1 imprints a reversible transcriptional programme of senescent cells
