Reprogramming lipid metabolism prevents effector T cell senescence and enhances tumor immunotherapy

https://doi.org/10.1126/scitranslmed.aaz6314

Senescence Reprogramming by TIMP1 Deficiency Promotes Prostate Cancer Metastasis

https://doi.org/10.1016/j.ccell.2020.10.012

Developmentally-programmed cellular senescence is conserved and widespread in zebrafish

https://doi.org/10.18632/aging.103968

The bright and dark side of extracellular vesicles in the senescence-associated secretory phenotype

https://doi.org/10.1016/j.mad.2020.111263

T cells with dysfunctional mitochondria induce multimorbidity and premature senescence

https://doi.org/10.1126/science.aax0860

Physiological hypoxia restrains the senescence-associated secretory phenotype via AMPK-mediated mTOR suppression

https://doi.org/10.1016/j.molcel.2021.03.018

Global hyperactivation of enhancers stabilizes human and mouse naive pluripotency through inhibition of CDK8/19 Mediator kinases

https://doi.org/10.1038/s41556-020-0573-1

H3K9me3-mediated epigenetic regulation of senescence in mice predicts outcome of lymphoma patients

https://doi.org/10.1038/s41467-020-17467-z

AP-1 imprints a reversible transcriptional programme of senescent cells

https://doi.org/10.1038/s41556-020-0529-5

Galacto-conjugation of Navitoclax as an efficient strategy to increase senolytic specificity and reduce platelet toxicity

https://doi.org/10.1111/acel.13142